cytokine storm
Interstitial Macrophages: Key Culprits in Severe COVID-19
The Coronavirus Exploits Interstitial Macrophages for Replication
From Cold to Pneumonia: The Role of Macrophages in Coronavirus
Activated Interstitial Macrophages as a Target for COVID-19 Treatment
COVID-19 Increases the Risk of Osteoporosis
Inflammatory Cytokines Trigger Bone Tissue Degradation
Type I Interferon Protects against Hyperinflammation and Tissue Damage
Interferon inhibits the production of enzymes by macrophages that destroy tissue structure
Cause of Mental Disorders in COVID-19
Inflammatory proteins disrupt hippocampal neurogenesis
Combined Interferon And Janus Kinase 1/2 Inhibitor In Treating COVID-19
Ruxolitinib and interferon resist cytokine storm and thrombosis; interferon inhibits viral replication
Senolytics Prevent Severe Coronavirus
Virus-induced cell senescence is a driving force and therapeutic target for COVID-19
Interferon Prevents Coronavirus Cytokine Storm in Diabetic Patients
Interferon increases levels of the enzyme SETDB2, which suppresses inflammation
The role of type I interferons in the development of severe COVID-19
Comparison of immune responses in mild and severe forms of COVID-19 and severe flu
Immunotherapy complication: cytokine release syndrome
Blocking the increasing production of catecholamines reduces inflammatory damage and mortality
COVID-19: Extrapulmonary Manifestations
The disease damages the blood-forming organs, blood vessels, kidneys, liver, gastrointestinal tract, skin, endocrine and nervous systems
The COVID-19 pandemic has increased the intensity of interferon studies
The research of interferon-alpha-2b for the treatment and prevention of COVID-19 has the most significant interest
Early interferon therapy reduces mortality in patients with COVID-19
A retrospective cohort multi-study
Studying COVID-19 to understand the immune hyperactivity
Pathological immune responses in COVID-19 lead to damage to blood vessels and internal organs. Severe clinical cases could be a source that allows us to better study disorders of immune regulation